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The after-effects are mediated by long-term potentiation (LTP)-like mechanisms where the upregulation of neurotransmitter release facilitates the opening of AMPARs and indirectly that of NMDARs (95)

For further reading on reconstitution math and handling, refer to educational guidelines at the National Center for Biotechnology Information

The degradation process of tryptophan in KP produces a variety of neuroactive metabolites with amino acid-like structures, such as an N-methyl-D-aspartate (NMDA) receptor agonist quinolinic acid (QA), and a neuroprotective NMDA receptor agonist kynurenic acid (KYNA)

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