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Although GSH does not directly reduce Ero1, it provides the electrons to recycle oxidized PDI, which, in turn, reduces Ero1 [160,169] (Fig

Therefore, regulation of intracellular GSH level is a promising strategy to boost the efficacy of antitumor treatment, as the exhaustion of antioxidants is highly desirable to sensitize cancer cells to various therapeutic means, such as chemotherapy, photodynamic therapy (PDT), chemodynamic therapy (CDT), and radiotherapy

doi: 10.1186/s13099-025-00755-8 150 SorianoA.HonoreP

Some clinical features of lead poisoning are similar to those of certain genetic mutations that result in pyrimidine excess due to enzyme deficiency