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Experimental data indicate that increased PER1 expression may enhance the susceptibility of tumor cells to ionizing radiation, potentially affecting thresholds for radiation-induced cell death in in vitro and in vivo systems

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2 Nonstandard administration of dose-reduced DOACs beyond the well-defined indications for dose-adjusted or low-intensity therapy can lead to subtherapeutic drug levels and reduced efficacy

The functional antagonism between Th17 cells and Treg cells primarily manifests in their reciprocal interactions and regulatory mechanisms within the immune system ( Treg cells mainly fall into four categories and use direct or indirect interaction to carry out immunosuppressive actions: 1) Cytokine-mediated suppression: TGF-, IL-10, and IL-35 induce Treg development, prevent antigen-presenting cell (APC) maturation, and mediate bystander suppression ( 3 Metabolic programs governing the Th17/Treg balance Metabolic reprogramming critically controls Th17 and Treg cell fate