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This study indicates that nicotine could be beneficial for conditions mediated by chronic neuroinflammation such that delineating the neural mechanism mediating these effects could lead to targeted therapeutics for both neuroinflammation and cognitive symptoms in PLWH

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Long-term use of e-cigarettes increases lung inflammation levels, including macrophages, neutrophils, eosinophils, and basophils, promotes the migration and infiltration of inflammatory cells from the blood to the respiratory tract, stimulates the production of Th2-related cytokines (IL-4, IL-5, IL-13), and exacerbates inflammation [40, 49]

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