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Clinically and mechanistically, PAH is distinguished by selective pathological remodeling of small distal pulmonary arteries, strong genetic susceptibility (e.g., bone morphogenetic protein type II receptor [BMPR2] gene mutations), and progressive pulmonary vascular dysfunction independent of left heart or parenchymal lung disease, making it the most extensively studied PH subtype in the context of molecular regulatory mechanisms (Ruopp and Cockrill 2022)

He is an assistant professor of neurology at Yale School of Medicine in New Haven, Connecticut