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Notch signaling as a mediator of ECE exposure-caused ciliated cell remodeling and cilia shortening Since Notch signaling activation is essential for airway epithelial cells differentiation in mice and humans [20, 26, 27], and Notch signaling strength was upregulated in ciliated cell subpopulations upon ECE treatment (Fig

To date the in-vivo occurrence of a persistent state(s) in Chlamydia resembling that observed in cell culture is consistent with morphological and clinical observations