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Recent reports of rapid onset changes in aortic stiffness after exposure to vape, of mice exposed to vape with nicotine developing features of chronic obstructive pulmonary disease (COPD), and the suppression of immune and inflammatory-response genes in nasal cells are examples of possible early causes for concern

While each of these methods can work in the short term, they often disappoint when it comes to heavy, long-term smoke damage

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