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Here, we report for the first time an antioxidant function for LDHB in protecting KRAS -mutant lung cancer from ferroptosis, a ROS-dependent non-apoptotic cell death program triggered by iron- dependent accumulation of lipid peroxides in plasma membranes and intracellular organelles such as mitochondria [31]
The treatment normalized neurotransmitter levels (norepinephrine, dopamine, serotonin), highlighting its potential in ameliorating AD-like symptoms ( In vivo , HJG shortened swim times in the Morris Water Maze and increased phosphorylated CREB levels in A-injected Wistar rats, demonstrating its neuroprotective potential (Kubota et al., 2017)
found that luteolin reduces uterine weight, exhibits pro-apoptotic and anti-proliferative effects, improves the proliferation of fibroid cells in a rat uterine fibroid model induced by diethylstilbestrol and progesterone, significantly lowers -SMA protein expression, and inhibits TGF-1 and PI3K-mediated signaling pathways by enhancing PTEN expression, ultimately achieving an anti-fibroid growth effect [129]
