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SFN is able to react with the thiol groups of Keap 1 and form thionacyl adducts promoting Nrf2 dissociation from Keap1 and subsequent activation of ARE driven gene expression and this may explain the increased GSH levels measured in control cells following SFN treatment

OPTN possesses a ubiquitin-binding domain that enables it to recognize and bind to polyubiquitinated cargoes, thus aiding their transportation for the formation of autophagosomes (Wong and Holzbaur, 2015)

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Inhibiting lipid peroxidation in bone microenvironment cells may represent a promising therapeutic approach for treating bone-related diseases