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Recent progress in transcriptomics, proteomics, and single-cell technologies is beginning to illuminate the heterogeneity and context-specific plasticity of astrocyte subtypes across disease states, offering an unprecedented opportunity to develop targeted interventions with cell-type and disease-stage selectivity

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When cancerous cells are generated, the expression of GPx1 is abnormal, causing intracellular ROS dysfunction [77, 78]

However, more research is needed to fully elucidate the mechanisms of action of MOTS-c and to translate these findings into therapeutic applications