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This review aims to summarize the role of the oxidative stress in the pathogenesis of AD, focusing in the main oxidative products (DNA, protein, and lipid oxidation products), as well as antioxidant therapeutic approaches involving natural products, nanomaterials, hydrogels, and microneedles
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published in the Journal of Molecular Medicine identified VEGFR2 (vascular endothelial growth factor receptor 2) activation as a central mechanism of the BPC-157 peptides pro-angiogenic effects
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