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During ischemia, neurons and glia undergo metabolic reprogramming toward FAO to compensate for glycolytic collapse, generating ROS and pro-inflammatory lipid mediators (e.g., arachidonic acid derivatives) that exacerbate neurovascular injury [253]

Although these findings appear promising, the utility of intravenous NAC repletion as a therapeutic strategy in PD is greatly limited by invasiveness and inconvenience of intravenous delivery

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The GPXs mediate the same reaction as catalase, namely, the recycling of hydrogen peroxide into water