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In studies using human fetal fibroblast cultures, Epithalon treatment extended cellular proliferative potential from termination at the 34th passage in control populations to beyond the 44th passage in treated cells, effectively surpassing the Hayflick limitthe maximum number of divisions normal cells can undergo before senescence

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have illustrated that ATX was able to restore GSH levels, clear ROS, reserve mitochondrial membrane potential, and structure damage in chondrocytes under the stimulation of IL-1

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