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BSO treatment alone did not significantly decrease cell viability (data not shown) and had no profound effect on the cytotoxicity of AQ or DEAQ ( Figure 2 )

This intricate relationship highlights CHAC1 as a pivotal player in the cellular response to oxidative stress, linking metabolic and stress response pathways in a manner that can critically influence cell fate under stress conditions ( 4.2 Unfolded protein response CHAC1 plays a crucial role in the UPR, specifically within the ATF4-ATF3-CHOP signaling pathway

43 , W154W159 (2015)