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Activated endothelium displays increased TF expression, triggering extrinsic and intrinsic coagulation cascades and contributing to a prothrombotic endovascular surface [80]

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Moreover, salt stress increases its cytotoxicity on biomolecules, and causes brutal oxidative damage to proteins either (i) directly through accelerating ROS production, or (ii) indirectly by the overproduction of advanced glycation end products (AGEs) 14,15

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