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Some are pathogenic themselves and cause their own set of symptoms, while others act alongside Borrelia burgdorferi to create a biofilm that makes them resistant to antibiotics

The incidence of these side effects tends to decrease with continued use and proper dose titration

The documented mechanisms are: VEGF stimulation in dermal fibroblasts that supports the microvascular network feeding the follicle, collagen and glycosaminoglycan synthesis at the follicular basement membrane, anti-inflammatory signaling that calms the chronic micro-inflammation associated with androgenetic alopecia, and direct follicular signaling that extends the anagen growth phase

Elevated peptide concentrations provide stronger inflammatory modulation, crucial for managing severe tissue damage and chronic inflammatory conditions