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We found that PA triggered a ferroptosis-like injury program in the ENS, marked by increased iron import, reduced iron export, elevated labile iron and mitochondrial ROS, GPX4 suppression, lipid peroxidation, and neuronal dysfunction

Overexpression of FSP1 or treatment with Fer-1, a ferroptosis inhibitor, significantly reversed the apoptosis, ferroptosis, and lipid peroxidation induced by the activation of the MPO/HOCl signaling pathway, exerting neuroprotective effects

Particles can be degraded peptide fragments, bacterial colonies, or contaminants introduced during handling

For example: Ginkgo biloba and ginseng may affect blood flow and have mild blood-thinning properties