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Under conventional interpretation, small monomeric or dimeric molecules activate Trks upon binding by inducing receptor dimerization

[ 49 ] Our study demonstrated that targeted loss of BACH1 in mouse cardiac tissue mitigates cardiac hypertrophy caused by Ang II and pressure overload, reduces cardiac fibrosis, and maintains cardiac function

This allows CHAC1 to post-translationally regulate protein activity by binding and cleaving specific -glutamyl amino acids ( Although CHAC2 enzymes in Leishmania major were shown to degrade only reduced and not oxidized glutathione, this specificity has yet to be confirmed for CHAC1 ( CHAC2 is both evolutionarily older and structurally distinct from CHAC1

Our flexible toolset is designed for people with homocystinuria to use with their caregivers and care teams to support the transition from childhood to adulthood