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It can easily oxidise, turning into "oxidized glutathione," which is no longer effective for neutralising free radicals

Recent investigations further indicated that UA may operate as a protective factor mediated through astroglia for dopaminergic neurons from glutamate toxicity ( + -induced depolarization amplifies neuroprotection provided by UA through a mechanism involving Ca 2 elevation and extracellular signal-regulated kinases (ERK 1/2 ) activation ( Figure 3 )

Inaba H, Pan D, Shin YH, Martin W, Buchman G, De Groot LJ

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