Taken together, these findings suggest that APR-246 could induce tumor cell death through both reactivation of mutant p53 and inhibition of cellular thiol-dependent redox system proteins (GPX4, TXNDR1, GRX1), providing novel combinatorial strategies for AML therapy
Molecular identification of carnosine synthase as ATP-grasp domain-containing protein 1 (ATPGD1)
21 Oral administration may be beneficial for patients with preclinical toxic effects or hepatic injury, although the presence of altered mental status and vomiting may limit its use
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