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Traumatic brain injury (TBI) is characterized by both the primary damage resulting from physical disruption of neural and vascular structures and the early emergence of secondary pathogenic events, which collectively contribute to neurologic deficits (Andriessen et al., 2010

doi: 10.1038/jcbfm.2010.29

Depression models: P21 shows antidepressant-like effects in animal models of depression, potentially through neurogenesis enhancement and GSK-3 inhibition

Chronic stress increases cortisol levels, which directly affects: Melanin production Hair growth cycles Scalp circulation Stress also accelerates oxidative damage, one of the main triggers of premature greying