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In distal TECs, activation of autophagy, such as upregulation of LC3-II/ATG7 and inhibition of AKT-mTOR, had anti-fibrotic effects, suggesting a protective role (Nam et al., 2019)

Optimal outcomes typically require consistent administration over months, allowing cellular glutathione stores to replenish and antioxidant defense systems to optimize

Understanding the mechanisms by which oxidative damage disrupts the BBB and facilitates the entry of blood-derived proteins into the brain is crucial for developing therapeutic strategies to mitigate BBB disruption, neuroinflammation, and neurodegeneration