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10.1016/S0140-6736(14)61393-3 92 Kalinowska-yszczarzA.SzczuciskiA.PawlakM

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This mechanism facilitates the controlled release of various therapeutic agents, including chemotherapeutics, photosensitizers, sonosensitizers, peptides, proteins, and nucleic acids

Persistent ferroptosis in alveolar and endothelial cells triggers pro-fibrotic signaling pathways, such as those involving TGF- and connective tissue growth factor (CTGF), which drive ECM deposition and fibroproliferation