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Mitochondrial GSH (which makes up 1015% of total cellular GSH) is the primary defense line protecting mitochondrial DNA and the electron transport chain from radical-induced degradation

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Moreover, the action of Semax, which is able to reduce the disturbances caused by ischemia, can be explained by joint action of peptide on receptors in an allosteric manner, together with hormones and mediators of the ischemic response

E3 ubiquitin ligase COP1 confers neuroprotection in cerebral ischemia/reperfusion injury via regulation of transcription factor C/EBP in microglia