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More recently, miR-141-3p, downregulated by lncRNA HIF1A-AS2, has been shown to promote CRC metabolic reprogramming and proliferation through FOXC1 activation, highlighting additional post-transcriptional networks under hypoxic stress [114]

While the discovery of therapeutic strategies targeting TICs holds promise, our understanding of TIC biology remains limited due to the challenges in identifying and isolating them

Affected individuals display a spectrum of disease severity corresponding to the degree of reduction in G6PD activity, which is determined by the specific G6PD variant alleles present 7,8,9