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MiD49 and MiD51 are chordate-specific mitochondrial elongation factor proteins, both of which can recruit Drp1 to the mitochondrial fission site independently of Fis1 and Mff and can also coordinate with Mff to regulate Drp1-mediated mitochondrial fission (Kraus and Ryan, 2017)

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The dual-mechanism approach may also offer specific advantages for particular patient subgroups, including those with type 2 diabetes and obesity where cardiometabolic improvements beyond weight loss are clinically important

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