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The level of OGD-induced ferroptosis in ADSCs was assessed according to the viability of cells treated with the ferroptosis inhibitor Ferrostatin-1 (Fer-1) 5 M, autophagy inhibitor 3-Methyladenine (3-MA) 5 mM, apoptosis inhibitor Z-VAD-FMK 10 M, necroptosis inhibitor Necrostatin-1 (Nec-1) 50 M, or ferroptosis activator erastin

Building on these findings, studies have shown that the active mitochondrial network in CRC cells exerts a physical metabolic occupancy effect that impedes immunological synapse formation, while the associated release of ROS further suppresses immune cell function

The treatment carries certain risks, such as allergic reactions, kidney or thyroid damage, infections, ochronosis, and toxicity

& Coto-Montes, A