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Administration of exogenous carnitine as an adjunct to VPA therapy is thought to restore beta-oxidation to mitochondria cells of the liver, accept toxic acyl moieties from CoA, and relieve the inhibition of urea synthesis.[3] However, our findings suggested that the effective utilization of levocarnitine transport into mitochondria, which may be individually different, might play an important role to lead these favorable processes

64 , 65 It is worth noting that increased studies have shown that many AMPs have significant selective anti-cancer activity 6668 and such AMPs can also be called anti-cancer peptides (ACPs)

The influence of endothelial metabolic reprogramming on the tumor microenvironment