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The other study used the stop-signal reaction time task and demonstrated causal relationships between cerebellar activity and error-related activation in the thalamus and the supplementary motor area, which in turn correlated with activation in the lateral prefrontal cortex during post-error slowing [345]

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Consistently, by ChIP assay, we detected significant NRF2 binding in Nqo1 and Slc7a11 loci, along with induced association of H3K4Me4 and H3K9Ac after DMF treatment (Fig