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When the central repressors of ferroptosis, such as glutathione (GSH) and lipid enzyme glutathione peroxidase 4 (GPX4) [103], which participate in peroxide reduction and reduce product toxicity, show insufficient activity, the lipid radicals formed cannot be cleared and continue to generate new oxidative products via chain reaction

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There is a growing recognition of the importance of glutathione (GSH) physiology and the number of physiological functions involving this small but important tripeptide

2018), and its elevated expression led to neuronal death and BBB destruction