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Roles of Hepatic Macrophages in Fatty Liver Disease Normally, KCs contribute to maintaining tissue homeostasis by expressing low levels of major histocompatibility complex II molecules and co-stimulatory molecules (45), high levels of programmed cell death ligand 1, and inhibitory cytokines IL-10 and TGF-

Importantly, NOX utilizes NADPH to produce superoxide, thus depleting NADPH needed by antioxidant systems such as thioredoxin reductase and glutathione peroxidase

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1 The management of T2DM is becoming increasingly complex with the availability of more pharmacologic drug classes and the completion of high-quality clinical trials