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This broad pathophysiological impact underscores the necessity of holistic therapeutic strategies targeting both urate-lowering and anti-inflammatory mechanisms

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This approach will delineate the relative contributions of ULK1-dependent versus -independent pathways under specific physiologic conditions, as well as elucidate the cell type-specificity of the crosstalk regulation between these pathways

Pro-inflammatory cytokines, including TNF-alpha, show reduced expression in BPC-157-treated models, limiting the duration of the acute inflammatory phase that delays tissue remodeling