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The direct contact of UCB with nerve cell membranes, which results in higher oxidative damage, the permeability of membranes, and reduced order in lipids and proteins, may be the cause of the potential disruption of cell equilibrium [20]

However, intracellular self-assemblies often lead to irreversible accumulation and end-effect cellular responses for therapeutic applications 21,22,23 , including mechanical stress, homeostasis disruption and eventual cell death

[DOI] [PMC free article] [PubMed] [Google Scholar] 12.Stern L, Gautier R II

1 Separately, GHK-Cus broad anti-inflammatory and antioxidant profile is relevant because chronic perifollicular micro-inflammation and oxidative stress are implicated in follicle decline