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The resolution of this discrepant event lies in the dependence of magnetic field strength as a function of the ratio of magnet length to radius

In addition, by screening differentially expressed genes (such as endothelial NOS and inflammatory factors) through transcriptomics, validating key proteins (such as VEGF and ICAM-1) through proteomics, and analyzing small molecule metabolites (such as NO and oxidative stress markers) through metabolomics, a molecular network of endothelial injury can be jointly constructed [33]

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Expression is normalized against ACT1 as reference gene