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Research confirms that in uterine fibroid tissue, the accumulation level of the oxidative damage marker 8-OHdG shows a significant positive correlation with MED12 mutation status, suggesting that an OS environment may represent a key selective pressure driving MED12 hotspot mutations [112]

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In CME_a, the glutamate portion of the CME forms stable chelation with the double copper binding site, and CME forms hydrogen bonds with H84, H262, and N259 in the pocket, which greatly contributes to the overall stability

(Also at risk: sterile forms of ALA, curcumin, vitamin B6, vasoactive peptide.) You are not required to cite specific regulatory text throughout your comment