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A critical role for DnaJ heat shock protein family (Hsp40) member C12 (DNAJC12) in breast cancer chemotherapy resistance was identified, as DNAJC12 was shown to suppress both DOX-induced ferroptosis and apoptosis, contributing to treatment resistance

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Finally, we found a similar decrease in survival when treating CCNE1-driven cells with PARP inhibitors that have less PARP-trapping activity than olaparib (Extended Data Fig