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Mechanisms such as microembolism, neurohormonal activation, exogenous and endogenous toxins, hemodynamics, inflammatory factors, I/R injury, and OS may be involved

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5o, p, r S2), indicating that GSH inhibited high glucose-induced PSC activation

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Glucagon-like peptide-1 receptor agonists decrease medical and surgical complications in morbidly obese patients undergoing primary TKA

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Hyperoside may help treat diabetic nephropathy by regulating the T cell balance towards anti-inflammatory Th2 and Treg populations and by promoting macrophage polarization from the M1 to the M2 phenotype (216)

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