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The Cs and nanospanlastics treated groups exhibited identical lesions to those observed in the GN-induced acute renal failure group, together with significant hydropic vacuolation of the renal tubular epithelium

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found that the compound ML316 can selectively inhibit the mitochondrial phosphate carrier Mir1 in fungi and then reduce mitochondrial oxygen consumption, leading to an unusual metabolic catastrophe in which ATP production is inhibited and fungal death [96]

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