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Before advancing these strategies to clinical application, several key challenges must be addressed, such as the complexity and functional redundancy of the ferroptosis regulatory network, the limitations of existing animal models in simulating the pathological features of human chronic OA, and the lack of reliable biomarkers for assessing intra-articular ferroptosis activity in humans

Most patients have a small but permanent decrease in eGFR without progression to end-stage renal disease requiring dialysis ( Table 2 ) (44, 52)

[ 13 ] ( Figure 6 ) Endothelial regeneration after injury reduces restenosis risk in coronary artery interventions