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Cluster 1 had severe systolic dysfunction, Cluster 2 had a relatively normal cardiac function, and Cluster 3 exhibited a hypertrophic phenotype (Image 1)

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Mechanistic studies reveal that high-fat diets elevate circulating BCAAs levels, activating the mTORC1 signaling pathway in skeletal muscle, thereby inducing lipid deposition and ultimately promoting obesity-associated insulin resistance (Newgard et al., 2009)

Moreover, pathogen-derived toxins enhance inflammatory chemokines or cytokines expression in response to infection by the host and can adversely affect BBB function [76]