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For IGF-1R/InsR HybridR activation, IGF-1R immunoprecipitates were probed with an antibody against the InsR specific pTyr 1334 residue (#44809G, Invitrogen/ThermoFisher) 52 , and total HybridR determined by immunoblotting with an Anti-InsR antibody (#3025)

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The role of ferroptosis in endometriosis has not yet been systematically examined

[25] In addition, in TBI and hypoxia models, NAC has repressed the development of secondary injury through the modulation of important molecular pathways like the nuclear factor erythroid 2related factor 2 pathway and the upregulation of GPX4, an important enzyme that suppresses ferroptosis