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These particular beneficial effects, i.e., the activation of collateral pathways, should occur along with the activated molecular pathways (Tkalevi et al., 2007

Glucose-dependent insulinotropic polypeptide (GIP) stimulation of pancreatic beta-cell survival is dependent upon phosphatidylinositol 3-kinase (PI3K)/protein kinase B (PKB) signaling, inactivation of the forkhead transcription factor Foxo1, and down-regulation of bax expression