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A single-arm clinical trial of a 48-hour intravenous N -acetylcysteine protocol for treatment of acetaminophen poisoning

It therefore appears that the reduction of A levels is insufficient to treat AD, and therefore calls into question the centrality of the amyloid hypothesis. Thus, we discuss the important biological functions of the A peptide, which could illuminate the less-than-satisfactory results of these anti-amyloid therapies

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Biological Psychiatry 91(5):449458 Mangas A et al (2018) Overexpression of kynurenic acid and 3-hydroxyanthranilic acid after rat traumatic brain injury