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The increased amount of SH groups damage may be critically important to the function of signal-transduction and transcription events that utilize proteins containing these reactive sites (Brandes et al ., 2009)

This framework may help explain the heterogeneous kinetic phenotypes of moxifloxacin-associated DILI plus SJS/TEN and suggests the potential value of time- and host-based risk stratification in ICI-treated populations

What we know Individuals with a certain genetic variation (polymorphism) in MTHFR may have an increased risk of developing high blood pressure, cardiovascular disease, and cancer

Science 338 , 120123 (2012)