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Its documented effects are primarily related to cellular repair, inflammation control, and vascular health

Activation of Akt1 is usually cytoprotective, such as its function during endothelial cell hypoxia (Somanath, Razorenova, Chen, & Byzova, 2006), and Akt1 has been extensively studied and is considered to be neuroprotective in stroke (Zhao, Sapolsky, & Steinberg, 2006), with Akt1 and Akt3 proteins degrading as early as 1 hr after stroke (Xie et al., 2013)

It is particularly important for liver function, as it helps to prevent the accumulation of fat in the liver and supports its detoxification processes

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