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Defects in mitochondrial dynamics disrupt the allocation of mtDNA throughout the network and subsequently lead to mitochondrial dysfunction and increased ROS

RA patients of this study were recruited at a university hospital based rheumatologic outpatient clinic

Treatment of cells with a peptide containing only the HIV-TAT sequence, as well as a peptide where the HIV-TAT sequence was introduced into the N-terminus of p53 TAD PEP, resulted in a minimal induction of cell death (Figure

This creates a destructive, self-perpetuating cycle where inflammation causes more damage, which in turn creates more inflammation and pain