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Thus, the use of this approach allowed to identify an original target, the protein kinase DYRK1B, suggesting that an inhibitor of this protein could present a possible senolytic activity 40

By enhancing angiogenesis: Nutrients and oxygen reach damaged tissues faster Inflammatory byproducts are cleared more efficiently Regeneration speed increases across soft tissue, joints, and muscle BPC-157 promotes angiogenesis and capillary density in damaged tissues through modulation of VEGF and endothelial nitric oxide synthase (eNOS). Sikiric et al., Regenerative Pharmacology 2

462 Inhibition of the NF-B signaling pathway leads to the decreased production of inflammatory mediators, including adhesion molecules and chemokines, which are crucial for the recruitment of immune cells to sites of inflammation