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Genomic studies and animal models have indicated that Se intake influences the expression of selenoprotein genes and pathways key to colorectal carcinogenesis such as the antioxidant response, immune and inflammatory pathways (including NF-kB and Nrf 2 signaling), the Wnt signaling pathway, protein synthesis pathway (Ye et al., 2021)

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Cancer cells dependence on glutamine could also serve as a basis for tailoring therapies to specific people