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We focus on delineating how this integrated approach moves beyond the limitations of conventional monotherapy, explore the emerging evidence supporting its clinical application, and identify key questions that must be addressed to advance its role in modern, precision-oriented UC management

When cholesterol levels suffice, SREBP-2 is inactive and located in the endoplasmic reticulum (ER) associated with SREBP-2 cleavage activation protein (SCAP) [17]

Though this is not an interaction in the traditional sense, it is an adjunctive measure that may improve tolerance of NAD+ therapy