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Superoxide dismutase protects cells exposed to oxygen, which can remove superoxide anion radicals in the body and effectively resist the damage of oxygen free radicals to organisms

There is no mention in in vivo studies of any notable risk associated with liposomal glutathione supplementation

however, emerging evidence indicates that alternative release pathways may also exist [193]

Cytoplasmic IDH1 and mitochondrial isocitrate dehydrogenase 2 (IDH2) reversibly synthesize -ketoglutarate from isocitrate and function in the reversible mitochondrial citrate--ketoglutarate shuttle (CAS) that shuttles NADPH reducing equivalents between the cytoplasm and the mitochondrial matrix (Figure 2D)